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ICAR AICE-JRF/SRF Veterinary Gynaecology & Reproductive Endocrinology: practice questions

ICAR AICE-JRF/SRF (PhD) · India · NTA for ICAR

Original practice questions written to the published syllabus · not past papers
10worked here, free
19written to this exam in this subject
11syllabus units

About ICAR AICE-JRF/SRF

ICAR AICE-JRF/SRF (PhD) is a PhD, fellowship and lectureship exam run by NTA for ICAR, for candidates in India. Computer-based, 2 hours, 120 questions: Part A 20 (general agriculture, animal husbandry, current events), Part B 50 (core group), Part C 50 (specialised subject). +4 / -1, maximum 480.

Source: icar.org.in.

Checked against the exam body's published information. Formats and fees change; confirm the current edition there before you book.

Veterinary Gynaecology & Reproductive Endocrinology in ICAR AICE-JRF/SRF

Deeper endocrinology: follicular waves, maternal recognition signals, luteolysis mechanism, cyst pathogenesis, Ovsynch variants and their rationale, buffalo reproductive biology, uterine disease classification.

The female reproductive tract, the oestrous cycle and its hormones, heat detection and synchronisation, infertility and pregnancy diagnosis, with emphasis on cows and buffaloes.

Our 19 ICAR AICE-JRF/SRF questions in this subject: 0 easy, 7 medium, 12 hard, across 11 units.

10 practice questions, worked

Choose an answer, then open the working. Each explanation says why the right answer is right and why the others are not.

These 10 were written to ICAR AICE-JRF/SRF and are not shown on any other exam's page.

Question 1 · hard

White heifer disease is best described as:

  1. XX/XY chimerism from placental vascular anastomosis with a male co-twin
  2. Segmental Müllerian duct aplasia linked to the white coat colour in Shorthorns
  3. Hereditary ovarian hypoplasia seen mainly in Swedish Highland (Mountain) cattle
  4. Persistent hymen caused by oestrogenic pasture
Answer and working

B. Segmental Müllerian duct aplasia linked to the white coat colour in Shorthorns

White heifer disease is a segmental aplasia of the Müllerian duct derivatives associated with the gene for white coat colour in Shorthorns; fluid often accumulates in front of the block. Chimerism describes freemartins and ovarian hypoplasia is a separate inherited condition.

Question 2 · medium

In cattle, the maternal recognition of pregnancy signal and its approximate timing are:

  1. Oestrogen from the conceptus, around days 11-12
  2. Chorionic gonadotrophin from endometrial cups, around day 40
  3. Interferon-tau, around days 15-17
  4. Relaxin from the placenta, around day 25
Answer and working

C. Interferon-tau, around days 15-17

Bovine trophoblast secretes interferon-tau around days 15-17, which suppresses endometrial oxytocin receptors and so prevents luteolytic PGF2α pulses. Conceptus oestrogen at days 11-12 is the pig's signal; endometrial-cup gonadotrophin is equine.

Question 3 · hard

Why does hysterectomy prolong the life of the corpus luteum in the ewe but not in the bitch?

  1. Ovine luteolysis needs uterine PGF2α; the canine CL regresses without a uterine signal
  2. The bitch's CL is refractory to PGF2α throughout dioestrus, so no uterine signal can act
  3. The ewe's CL depends on uterine prolactin
  4. Hysterectomy removes the source of LH in the bitch
Answer and working

A. Ovine luteolysis needs uterine PGF2α; the canine CL regresses without a uterine signal

In ruminants, sows and mares the uterus produces the luteolytic PGF2α, so removing it maintains the CL. The canine CL has a set lifespan independent of the uterus, which is why pregnant and non-pregnant dioestrus last about the same time.

Question 4 · hard

Why do many buffaloes that are cycling in winter show anoestrus or silent heat in summer in northern India?

  1. Buffaloes are long-day breeders whose cycles are suppressed by the short nights of summer
  2. High-quality summer green fodder causes luteal cysts that block oestrus
  3. Heat stress with photoperiodic effects lowers oestradiol output and oestrous expression
  4. The buffalo CL becomes refractory to luteolysis in summer
Answer and working

C. Heat stress with photoperiodic effects lowers oestradiol output and oestrous expression

Buffaloes conceive mostly in autumn and winter; in summer, poor heat dissipation (dark skin, few sweat glands) and seasonal photoperiod effects suppress follicular oestradiol and oestrous behaviour. They behave more like short-day breeders, not long-day breeders.

Question 5 · medium

In a Presynch-Ovsynch programme, PGF2α is given on days 0 and 14. On which day is the first GnRH of Ovsynch given?

  1. Day 26
  2. Day 21
  3. Day 28
  4. Day 35
Answer and working

A. Day 26

Ovsynch starts about 12 days after the second PGF2α: 14 + 12 = 26. Repeating the 14-day gap gives day 28, adding a 21-day cycle to the second PGF2α gives day 35, and counting 21 days from the first PGF2α gives day 21. The aim is to begin Ovsynch between about days 5 and 12 of the cycle, when the first GnRH is most likely to ovulate a dominant follicle.

Question 6 · hard

The main role of kisspeptin in female reproduction is:

  1. Stimulating GnRH neurons and integrating steroid feedback and metabolic signals
  2. Directly causing luteolysis of the ruminant CL alongside uterine prostaglandin
  3. Acting as the conceptus signal for maternal recognition of pregnancy in the mare and jenny
  4. Inhibiting FSH secretion from the pituitary
Answer and working

A. Stimulating GnRH neurons and integrating steroid feedback and metabolic signals

Kisspeptin neurons in the hypothalamus drive GnRH secretion and mediate oestradiol feedback and energy-status effects, making them the gate of puberty and of the LH surge. Inhibin, not kisspeptin, suppresses FSH.

Question 7 · hard

Negative energy balance delays the first postpartum ovulation in dairy cows mainly by:

  1. Reducing LH pulse frequency, so dominant follicles make too little oestradiol for an LH surge
  2. Blocking all follicular wave emergence until body condition has fully recovered to calving level
  3. Causing premature luteolysis of the first CL
  4. Increasing FSH so that too many follicles ovulate
Answer and working

A. Reducing LH pulse frequency, so dominant follicles make too little oestradiol for an LH surge

In negative energy balance, low insulin and IGF-I and reduced GnRH/LH pulsatility mean follicular waves continue but dominant follicles do not reach ovulatory oestradiol output. Waves do not stop.

Question 8 · hard

The rationale for giving GnRH or hCG about 5-7 days after insemination in repeat breeders is to:

  1. Induce ovulation of the follicle that was inseminated, which in repeat breeders ovulates days late
  2. Cause luteolysis of a persistent CL so that the cow can return to oestrus sooner
  3. Form an accessory CL from the first-wave dominant follicle, raising progesterone for the embryo
  4. Stimulate uterine contractions to move the embryo
Answer and working

C. Form an accessory CL from the first-wave dominant follicle, raising progesterone for the embryo

An accessory CL raises progesterone in the period of embryo elongation and interferon-tau secretion; reported gains in conception are modest and inconsistent. The inseminated follicle has already ovulated by day 5.

Question 9 · medium

Which bacterium is most strongly associated with persistent clinical endometritis in dairy cows?

  1. Trueperella pyogenes
  2. Streptococcus equi subsp. zooepidemicus
  3. Taylorella equigenitalis
  4. Brucella canis (canine brucellosis)
Answer and working

A. Trueperella pyogenes

Trueperella pyogenes, often with anaerobes such as Fusobacterium necrophorum and Prevotella, is linked with persistent bovine endometritis. S. zooepidemicus and Taylorella are equine uterine pathogens.

Question 10 · hard

When sorting individual problem cows during a herd fertility visit, the three practical groups are:

  1. Heifers; cows; bulls
  2. High yield; medium yield; low yield
  3. Pregnant; aborted; culled
  4. Not seen in heat; bred but not conceiving; abnormal discharge
Answer and working

D. Not seen in heat; bred but not conceiving; abnormal discharge

Grouping by presenting problem directs the examination: anoestrus/silent heat/cysts, repeat breeding/subclinical endometritis, and metritis/endometritis/pyometra respectively.

A study plan for Veterinary Gynaecology & Reproductive Endocrinology in ICAR AICE-JRF/SRF

This plan follows where our questions actually fall, so the time goes where the marks are likely to be. It is our reading of the syllabus, not the exam body's weighting.

  1. First, the heavy units (58% of the 19 questions): Female reproductive physiology (4, 21%); Reproductive hormones (3, 16%); Cystic ovarian disease (2, 11%); Infertility in cows and buffaloes (2, 11%).

    Read the manual chapters for these first and do every worked question you can find on them.

  2. Then the middle band: Oestrus synchronisation protocols (2, 11%); Anoestrus (1, 5%); Endometritis (1, 5%); Female reproductive anatomy (1, 5%).

    Aim for one sitting per unit, then mixed questions across them.

  3. Then the lighter units: Pregnancy diagnosis (1, 5%); Repeat breeding (1, 5%); The oestrous cycle (1, 5%).

    Learn the definitions, classifications and standard numbers; these units are more often recalled than worked.

  4. Leave time for the hard ones: Anoestrus (1 hard of 1), Female reproductive anatomy (1 hard of 1), Pregnancy diagnosis (1 hard of 1).

    Come back to these after a first pass, untimed, and read every explanation, including for the options you rejected.

  5. Finish timed. Sit the subject as a timed set without notes, mark it, and send each wrong answer back to its unit above.

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