Australasian Veterinary Examination (AVE), formerly the National Veterinary Examination (NVE) · Australia / New Zealand · Australasian Veterinary Boards Council (AVBC)
Original practice questions written to the published syllabus · not past papersAustralasian Veterinary Examination (AVE), formerly the National Veterinary Examination (NVE) is a licensing and registration exam run by Australasian Veterinary Boards Council (AVBC), for candidates in Australia / New Zealand. Step 2, preliminary MCQ exam: computer-adaptive, Rasch-scored, cut-score 500. Paper 1 (base knowledge) 80 Q in 120 min; Paper 2 (clinical reasoning) 80 Q in 150 min. Held once a year in April (next: 16 Apr 2027). Step 3, clinical exam: 5 days at the University of Queensland, usually November.
Source: avbc.asn.au.
Checked against the exam body's published information. Formats and fees change; confirm the current edition there before you book.
Growing, conserving and grazing the forage that feeds ruminants: fodder crops, hay and silage, pasture management and toxic plants.
Our 16 Australasian Veterinary Examination, formerly the National Veterinary Examination questions in this subject: 1 easy, 8 medium, 7 hard, across 5 units.
Choose an answer, then open the working. Each explanation says why the right answer is right and why the others are not.
These 10 were written to Australasian Veterinary Examination, formerly the National Veterinary Examination and are not shown on any other exam's page.
Which temperate legume spreads by stolons and is the classic grazing partner of perennial ryegrass in New Zealand and north-west Europe?
A. White clover (Trifolium repens)
White clover's creeping stolons let it persist under close grazing alongside ryegrass. Red clover is a short-lived upright hay and silage legume; subterranean clover is the Mediterranean-climate annual of southern Australia; lucerne is upright and suffers under continuous close grazing.
Sheep grazing a pure Brachiaria decumbens pasture develop jaundice and dermatitis of unpigmented skin. What is the mechanism?
C. Saponin crystals in bile ducts block phylloerythrin excretion
B. decumbens saponins cause crystal-associated cholangiopathy; phylloerythrin from chlorophyll is not excreted in bile and reaches the skin, causing hepatogenous photosensitisation with jaundice. Brachiaria is not cyanogenic; nitrate causes methaemoglobinaemia, not photosensitisation; hypericin causes primary photosensitisation from St John's wort.
Which pair correctly matches a temperate grass endophyte alkaloid with its disease?
D. Ergovaline in tall fescue - fescue toxicosis
Ergovaline from the tall fescue endophyte causes fescue toxicosis; lolitrem B from the ryegrass endophyte causes ryegrass staggers. Facial eczema is caused by sporidesmin from Pithomyces chartarum, a litter fungus, not an endophyte; kale anaemia comes from S-methylcysteine sulphoxide in brassicas.
Several ewes fed poorly fermented, soil-contaminated big-bale silage develop facial paralysis and circle to one side. Which organism and silage defect are most likely involved?
C. Listeria monocytogenes, multiplying in silage with high pH and soil contamination
Listeria grows in silage that has failed to acidify (pH above about 5) and in soil-contaminated surface layers; it causes encephalitis with circling and unilateral facial paralysis. Tetanus, salmonellosis and pasteurellosis are not the classic silage-associated encephalitis.
A farmer believes that resting each paddock for 30 days between grazings will 'starve out' all infective nematode larvae. What is the correct advice?
D. Larvae can survive weeks to months on pasture, so a 30-day rest does not clean it
Free-living third-stage larvae survive for weeks to months depending on temperature and moisture, so short rest periods do not sterilise pasture. They do not die in 48 hours, survival is not tied to fertiliser, and harrowing spreads rather than removes larvae.
Cattle grazing bracken-infested hill pasture for many months develop haematuria with bladder tumours. Which bracken toxin is responsible?
D. Ptaquiloside
Ptaquiloside is the carcinogen behind enzootic haematuria and acute marrow damage in cattle. Bracken thiaminase matters mainly in horses and pigs; lolitrem B is a ryegrass endophyte toxin; mimosine is from leucaena.
Pregnant mares grazing endophyte-infected tall fescue are at risk of which syndrome?
B. Prolonged gestation and agalactia
Ergovaline from the fescue endophyte suppresses prolactin and causes prolonged gestation, thickened placentas, dystocia and agalactia in mares. Haematuria is bracken; goitre and alopecia are mimosine/DHP; Heinz-body anaemia is kale (SMCO) or onion.
Horses kept for many months on pasture of high-oxalate tropical grasses such as setaria or buffel develop shifting lameness and swollen facial bones. What is the condition?
D. Nutritional secondary hyperparathyroidism ('big head')
Oxalate binds dietary calcium, so parathyroid hormone mobilises bone, producing fibrous osteodystrophy of the facial bones. Hypericin causes skin lesions; grass sickness is a dysautonomia; bracken thiaminase causes thiamine deficiency, not laminitis.
'Clover disease', with infertility of ewes grazing certain subterranean clover pastures in Western Australia, was caused by:
D. Phyto-oestrogens such as formononetin
Formononetin and related isoflavones act as oestrogens and cause temporary and permanent ewe infertility; low-oestrogen cultivars were bred in response. Dicoumarol causes haemorrhage, pyrrolizidine alkaloids cause liver disease and cyanogenic glycosides cause cyanide poisoning.
Cattle fed large amounts of kale develop haemolytic anaemia with Heinz bodies. Which compound is responsible?
C. S-methylcysteine sulphoxide
S-methylcysteine sulphoxide is converted in the rumen to dimethyl disulphide, which oxidises haemoglobin and causes Heinz-body haemolytic anaemia. Glucosinolates in brassicas are goitrogenic; lolitrem B and ergovaline are endophyte toxins of grasses.
This plan follows where our questions actually fall, so the time goes where the marks are likely to be. It is our reading of the syllabus, not the exam body's weighting.
Read the manual chapters for these first and do every worked question you can find on them.
Aim for one sitting per unit, then mixed questions across them.
Learn the definitions, classifications and standard numbers; these units are more often recalled than worked.
Come back to these after a first pass, untimed, and read every explanation, including for the options you rejected.
Do these early in revision so they are fast on the day.
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